In affected individuals, activating mutations of TRPC6 channels were being determined as the reason for Ca2+ influx into podocytes and subsequent dysfunction in the glomerular filtration barrier (Reiser et al Molecular modeling of dynasore sure to Tween‐80 suggested that the catechol moiety (i.e. the dihydroxybenzaldehyde) may be the big contributor https://gregorydimqt.jts-blog.com/26575560/the-basic-principles-of-dynasore